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The former hasno normal allele to offset the deleterious effects of a newmutation, while the latter are able to survive as long as asingle normal gene can provide sufficient normal protein forcell survival.

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Sample size calculations can be performed using theexpected allele frequency and assuming modest odds (or hazards) ratios (e.g.see Figure 7.3).

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Cholinergic activators Since brain AChlevels are markedly reduced and cholinergicneurotransmission is the major sufferer in AD,various approaches to augment brain ACh havebeen tried. High-magnification photomicrograph of a similar bone spicule stainedwith H&E, included for orientation purposes. Lin S et al (2011) Mutant p53 disrupts role of ShcA protein in balancing Smad protein-dependent and -independent signaling activity of transforming growth factor-? (TGF-?). Spinal osteomyelitis: a review of 10 years’ experience. Dose-dependent toxicity startsfrom the upper part of therapeutic concentrationrange (Fig.

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